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- Guillain-barré Syndrome after Multiple Bee Stings
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Sang-Chan Jin
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J Korean Soc Clin Toxicol. 2018;16(1):57-59. Published online June 30, 2018
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DOI: https://doi.org/10.22537/jksct.2018.16.1.57
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Abstract
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- Severe systemic responses including neurologic complications such as myasthenia gravis, myeloradiculopathy, optic neuropathy, parkinsonism, stroke and Guillain-$barr{acute{e}}$ syndrome can occur after bee stings. This case describes a 78-year-old female who presented with symptoms of acute progressive bilateral symmetrical weakness in both lower legs after multiple bee stings. Nerve conduction study findings were consistent with acute sensorimotor axonal neuropathy and recovered by treatment with intravenous immunoglobulin. This case highlights that bee stings can result in acute onset Guillain-$barr{acute{e}}$ syndrome, although the pathophysiologies of bee venoms need to be investigated accurately.
- Parkinsonism after Cyanide Intoxication: A Case Report
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Joo-Hyun Suh, Eun-Kyung Eo
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J Korean Soc Clin Toxicol. 2004;2(2):137-140. Published online December 31, 2004
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Abstract
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- Acute cyanide poisoning is usually the result of attempted suicide which is often lethal within minutes or leads to a very poor prognosis after delayed and inadequate treatment. It affects the cerebral structures with the highest oxygen requirement, such as the basal ganglia, the cerebral cortex. We experienced a-45-year-old man who ingested Potassium Cyanide. He was stuporous. In 25 minutes, respiratory arrest developed and cardiopulmonary resuscitation was done. After return of spontaneous circulation, he admitted to intensive care unit, and conservative treatment was started. The clinical status was improved by degrees, but he couldn't perform daily activity like before. Minimal limitation of movement and memory deficit were left. In magnetic resonance imaging, which taken at the 11th day after admission, there were both basal ganglia and folia of cerebellum abnormality.